Is carbidopa in the drug you are taking? There is a new modern approach to taking L-dopa without the devastating side effects.
As you know, Parkinson’s is thought to be a progressive neurodegenerative disease. This means that the disease progresses as more and more postsynaptic dopamine neurons in the brain are destroyed.
Our clinic have mastered using L-dopa without carbidopa.
The ideal scenario is to catch the disease as early as possible so that full function can be restored. This can be accomplished with amino acid therapy but only when there is enough dopaminergic neurons remaining to compensate for the damaged ones. No amount of amino acids will be able to completely restore proper function if too many dopaminergic neurons are destroyed.
Unfortunately, more often than not by the time a person is ready to try amino acids therapy damage is already done and the disease is in full force. While these people will still benefit greatly from amino acid therapy, most do not achieve complete relief of symptoms due to the extent of the neurodegeneration.
Therefore, if you are a victim of Parkinson’s disease you should consider properly guided amino acid therapy as soon as possible to obtain optimal results.
The damaged postsynaptic dopamine neurons in the brain cause a gradual decline in dopamine production and function. L-dopa is required in order to raise dopamine levels high enough to alleviate symptoms. The conventional medical standard of care for Parkinson’s disease focuses on symptomatic relief using L-dopa as a pharmaceutical drug to increase dopamine levels in the brain.
L-dopa can cross the blood-brain-barrier and is freely synthesized into dopamine if the necessary co-factors are present.
However, when given as a stand-alone therapy, L-dopa is associated with numerous side effects due to the fact that L-dopa used alone may deplete L-tyrosine, L-tryptophan, 5-hydroxytriptophan (5-HTP), serotonin and sulfur amino acids. Especially that Parkinson’s disease itself is associated with a depletion of tyrosine hydroxylase which is the enzyme responsible for catalyzing the conversion of L-tyrosine into L-dopa, dopamine, serotonin and norepinephrine.
Carbidopa is often given along with L-dopa in an effort to relieve some of the side effects of L-dopa. Carbidopa is a general decarboxylase inhibitor, which means it inhibits the peripheral synthesis of serotonin, dopamine, norepinephrine and epinephrine.
CARBIDOPA: The drug depletes vitamin B6 and accelerates Parkinson’s disease symptoms
The only reason for carbidopa use is to control L-dopa-induced nausea, it is of no benefit in controlling Parkinson’s disease symptoms.
Although this approach may relieve some of the side effects of L-dopa especially nausea, it is associated with a long list of often severe side effects and has been implicated as being a cause of Parkinson’s disease progression.
What if There Was a Way to Get the Great Results of L-dopa Without the Unpleasant Sometimes Severe Side Effects?
Well, There Is.
This approach is not a new form of therapy. It uses the most effective therapy available, L-dopa, then nutritionally controls all the major problems which have interfered with optimal administration of L-dopa in the past.
These problems include L-dopa-induced nausea, irreversible dyskinesias, on/off effect, and L-dopa that quits working.
This approach also effectively deals with the nutritional collapse of dopamine precursors (nutrients) that causes all symptoms related to Parkinson’s disease.
This approach does not use carbidopa which irreversibly depletes vitamin B6, the vitamin B6 enzymes and interferes with the function of over 300 other enzymes and proteins.
This approach was developed by a team of research lead by Mary Hinz, MD and is based on a medical database with over 3 million patient-days of treatment experience documents.
This research project has been co-published in peer-reviewed scientific papers with faculty of the University of Minnesota Medical School-Duluth, MN. At present 21 papers have been published.
The approach has found that all the side effects and adverse reactions associated with Parkinson’s disease along with treatment with L-dopa/carbidopa are caused by relative nutritional deficiencies. The success of this approach depends of the complex task of determining the proper balance of dopamine precursor nutrients and other nutrients required to return the system to normal function, a state where no side effects occur.
When the nutrient precursors of dopamine (L-tyrosine and L-dopa) are administered in proper balance with the other nutrients affected there is no side effects. Nutrients when given properly have no side effects.
The long term success of this approach can only be accomplished by working with a provider who is trained in the proper use of amino acid therapy. You can start this therapy today and begin your journey to a new life with our medically based nutritional protocol clinically proven to eliminate symptoms of Parkinson’s disease. GET STARTED
We are here to help you to have a better life with better health outcomes and we expect your regular doctor is in it for you as well.
It is well known in healthcare that vitamin B6 deficiency is associated with an increased death rate. Since Carbidopa was introduced in 1975 the Parkinson death rate has increased by 328% (Center for Disease Control). In order for carbidopa to work it has to induce a vitamin B6 deficiency. For person with Parkinson’s vitamin B6 deficiency symptoms can be exactly the same as symptoms of Parkinson’s disease. Vitamin B6 depletion can accelerate Parkinson’s disease. Vitamin B6 depletion can collapse dopamine synthesis. Vitamin B6 depletion can cause dyskinesias which appear to be irreversible if not corrected. Vitamin B6 depletion affects both the brain and peripheral system. Vitamin B6 deficiency can accelerate the collapse and deterioration of Parkinson’s disease. The only way to properly manage these problems is to not use carbidopa.
Our clinics use L-dopa properly, we do not:
- Facilitate Parkinson progression and collapse Induce nutritional deficiencies which have the same symptoms as Parkinson’s disease
- Use methods linked to an increased death rate
- Leave nutritional deficiencies untreated
- Induce irreversible dyskinesias
- Induce untreatable on/off effect
- Induce vitamin B6 deficiency which may compromise over 300 enzymes and proteins in the body.
To find out how we can help you, please contact our office at