We didn’t invent the wheel we just improved “This is not a new treatment! It is a nutritional improvement in side effect management of the most effective treatment available, L-dopa.”
From: Dr. Marty Hinz MD
The approach for optimal administration of a nutrient is different from a drug. The irreversible postsynaptic dopamine neuron damage at the heart of Parkinson’s disease induces a nutritional deficiency (a relative nutritional deficiency) of the dopamine precursors L-tyrosine and L-dopa. This nutritional fact is ignored by the standard of care in medicine who insists on prescribing L-dopa as a drug. Like hammering a square peg into a round hole, the process that gives no thought to the other systems being compromised by the standard of care method for administering L-dopa.
Herein lies the primary relative nutritional deficiency (RND) of Parkinson’s disease. The availability of nutrient dopamine amino acid precursors (L-tyrosine and L-dopa) determine the severity of symptoms experienced. Prior to onset of Parkinson’s disease symptoms adequate dopamine precursors were obtained from the diet, there is no symptoms, and no nutritional deficiency. With onset of symptoms nutritional intake of precursors is no longer adequate,which is the very definition of a relative nutritional deficiency. While the etiology of Parkinson’s disease is postsynaptic dopamine neuron damage, once this damage occurs it causes symptoms driven by the dopamine precursor relative nutritional deficiency it induces.
Increasing synaptic dopamine concentrations with L-tyrosine and L-dopa is like a turning up the voltage. More electricity flows through the compromised postsynaptic dopamine neurons restoring fine motor control and alleviating Parkinson’s disease symptoms.
All Parkinson’s disease symptoms represents dopamine precursor induced relative nutritional deficiency. If all of L-dopa side effects are controlled by proper administration of nutrients then it is asserted that L-dopa nutrients needs to be started first. Failure to do so is ignoring a nutritional deficiencies which are causing Parkinson’s disease symptoms and inducing collapse of other systems. The current standard of care in medicine neglects these nutritional deficiencies. The progression of the disease is a function of nutritional deficiency.
From one of Marty Hinz MD research papers: The hypothesis, “If drugs have side effects, nutrients and amino acids normally found in the system have no side effects when properly administered then when nutrients or amino acids normally found in the system displays side effects they have been converted to a drug through improper administration.”
Parkinson’s disease patients treated under this approach early enough in their disease have no symptoms of disease when stabilized. Failure to implement this approach leads to progressive irreversible post-synaptic neuron damage due to nutritional collapse of glutathione and other systems involved. Once damage progresses beyond a certain point symptoms can never be fully controlled even with optimal treatment. All too often we see patients drop out of care under this approach then go to a doctor that will write them prescription drugs while oblivious to the numerous nutritional collapses that are driving progression of the disease. Then after 12 to 18 months the patient returns with symptoms that are markedly worse. The problem is that more irreversible damage from progressive nutritional collapse has occurred due to nutritional neglect. Make no mistake the current standard of care in medicine contributes greatly to the neurodegeneration experienced by the Parkinson’s patient.
Patients need to start this approach as soon as possible not when the disease gets bad enough. The patient who waits to start this approach is essentially saying, “When my brain damage is bad enough and out of control enough I will do something about it.” Every day that proper nutrition is not given to the Parkinson’s disease patients is another day that irreversible system collapse occurs.
Parkinson’s disease is a classified as a “progressive neurodegenerative disease” which it indeed is when the patient is placed in the hands of mainstream medicine which is intent on writing drug prescription, doing surgery, and putting implants in the brains of people who are in reality suffering from a complex set of nutritional deficiencies (RND) at the heart of which is a relative nutritional deficiency of dopamine nutrient precursors.
ALL THE PROBLEM ARE NUTRITIONALLY BASED
The following are the major problems of Parkinson’s disease and it treatment with L-dopa/carbidopa. Every one of these problems represents a relative nutritional deficiency.
Parkinson’s disease is associated with “total loss of glutathione(GSH) and the other thiols L-methionine (Met), S-adenosyl-L-methionine (SAM), cystathione, and L-cysteine (Cys). Depletion of serotonin (5-HT), dopamine (DA), norepinephrine (NE), epinephrine (EPI), L-tyrosine (Tyr), and L-tryptophan (Try) is also known.
L-dopa induces nausea, new onset depression, on/off effect, and is prone to tachyphylaxis. L-dopa also is known to induce serotonin, thiols, L-tyrosine, and L-tryptophan related RNDs.
Carbidopa (not L-dopa) induces irreversibly dyskinesias and is linked to the increasing mortality rate due to induction of vitamin B6 (PLP) RND carbidopa irreversibly binding with vitamin B6 and the PLP-dependent enzymes which in turn causes drug-induced RND depletion and dysfunction of over 300 enzymes and proteins in the body. Carbidopa has almost 100 other side effects that it can induce published. When proper nutritional care is given carbidopa is not needed.
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